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Sally J. Pyle
Director -- Dr. Sally Pyle
(701) 777-3302 (Robertson/Sayre - Office 9)
BS, 1977 & MS, 1988 Iowa State University Ph.D., 1992 Duke University
Postdoctoral Fellowship, Pathology Departments, Brigham & Women's Hospital, Children's Hospital and Harvard Medical School, 1992-1994
Johnson & Johnson Toxicology Scholar Rutgers University, 1994-1997
Research Interests:Developmental Neurobiology, Neurotoxicology, Interactions of the Cytoskeleton
Jurgens, CWD, Boese, SJ, King, JD, Pyle, SJ, Porter, JE and Doze, VA. "Adrenergic receptor modulation of Hippocampal CA3 network activity," Epilepsy Res., 66:117-28, 2005.
Pyle, SJ, Roberts, KG and Reuhl, KR. "Delayed expression of the NFH subunit in differentiating P19 cells," Dev. Brain Res., 132(1):103-106, 2001.
Pyle, SJ and Reuhl, KR. Cytoskeletal Elements in Neurotoxicity. In: Comprehensive Toxicology. Vol. 11. Eds.: Sipes, I.G., McQueen, C.A., Gandolfi, A.J., Elsevier Science, Ltd., Cambridge, pp. 79-97, 1997.
Graham, DG, Amarnath, V, Valentine, WM, Pyle, SJ and Anthony, DC. Pathogenetic studies of hexane and carbon disulfide neurotoxicity. Crit. Rev. Toxicol. 25:91-112, 1995.
Pyle, SJ, Graham, DG and Anthony, DC. Dimethylhexanedione impairs the movement of neurofilament protein subunits, NFM and NFL, in the optic system. Neurotoxicology 15:279-286, 1994.
Pyle, SJ, Amarnath, V, Graham, DG and Anthony, DC. Decreased levels of the high molecular weight subunit of neurofilaments and accelerated neurofilament transport during the recovery phase of 2,5-hexanedione exposure. Cell Motil. Cytoskel. 26:133-143, 1993.
Pyle, SJ, Amarnath, V, Graham, DG and Anthony, DC. The role of pyrrole formation in the alteration of neurofilament transport induced during exposure to 2,5-hexanedione. J. Neuropathol. Exp. Neurol. 51:451-458, 1992.